Radiology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Sigel, B.
Right arrow Articles by Justin, J. R.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Sigel, B.
Right arrow Articles by Justin, J. R.

Radiology, Vol 148, 799-802, Copyright © 1983 by Radiological Society of North America

ARTICLES

Red cell aggregation as a cause of blood-flow echogenicity

B Sigel, J Machi, JC Beitler and JR Justin

The role of red cell aggregation as a cause of ultrasonic echogenicity in flowing blood was evaluated by in vitro experiments using fresh human blood. Blood was circulated in tubes of varying diameter (12 mm to 6 mm). In all experiments, echogenicity increased as blood approached static conditions. Echogenicity was greater in tubes with a larger diameter over the same range of blood velocity. However, echogenicity in tubes of various diameters was the same when evaluated in terms of shear rate. Thus, shear rate and not velocity is the flow condition that determines echogenicity. Since shear rate determines the degree of red cell aggregation, while other conditions affecting red cell aggregation (hematocrit, erythrocyte membrane conditions, and plasma macromolecules) are held constant, we conclude that these results provide additional evidence that red cell aggregation is a cause of echogenicity in flowing blood. Furthermore, a red cell aggregation mechanism for blood-flow echogenicity would explain the increased prominence of internal echoes in lower shear rate venous blood flow compared with those of higher shear rate arterial blood flow.


This article has been cited by other articles:


Home page
 ChestHome page
N. Shinokawa, T. Hirai, S. Takashima, T. Kameyama, K. Nakagawa, H. Asanoi, and H. Inoue
A Transesophageal Echocardiographic Study on Risk Factors for Stroke in Elderly Patients With Atrial Fibrillation : A Comparison With Younger Patients
Chest, September 1, 2001; 120(3): 840 - 846.
[Abstract] [Full Text] [PDF]

Home page
 CLIN APPL THROMB HEMOSTHome page
G. Caimi and L. Messina
Erythrocyte Aggregation in Nonvalvular Atrial Fibrillation
Clinical and Applied Thrombosis/Hemostasis, January 1, 2001; 7(1): 75 - 75.
[PDF]

Home page
 RadiologyHome page
J. M. Rubin, J. B. Fowlkes, T. A. Tuthill, A. P. Moskalik, R. T. Rhee, R. S. Adler, S. N. Kazanjian, and P. L. Carson
Speckle Decorrelation Flow Measurement with B-Mode US of Contrast Agent Flow in a Phantom and in Rabbit Kidney
Radiology, November 1, 1999; 213(2): 429 - 437.
[Abstract] [Full Text]

Home page
 Vasc MedHome page
P. C. Smith
Noninvasive venous investigation
Vascular Medicine, September 1, 1990; 1(2): 139 - 166.
[PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
RADIOLOGY RADIOGRAPHICS RSNA JOURNALS ONLINE
Copyright © 1983 by the Radiological Society of North America.